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Mark Your Calendars for Neuroscience 2022 Advocacy Events Neuroscience 2022 in San Diego is quickly approaching, and there is an exciting lineup of advocacy events to attend! Visit the ECPA poster session, where past and current ECPAs will be presenting on their policy and advocacy work, onSaturday, November 12 from 6:30–8:30 p.m. PSTin the San Diego Convention Center (SDCC) Poster Hall. The Animals in Research Panel will be held onMonday, November 14 from 1:30–3:30 p.m. PSTin the SDCC Room 11. Panelists will discuss the benefits and limitations of alternatives to animals in research and why neuroscience still needs animal models. Attend the Advocacy Forum onTuesday, November 15 from 2–3:30 p.m. PST, also in the SDCC Room 11, where panelists will explain how to successfully leverage the use of data and emotion to influence the decision-making process and have productive and impactful advocacy discussions. Please email the SfN Advocacy Team with any questions. ECPA Program Applications Open November 29 Appl...
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Meet two of current Early Career Policy Ambassadors (ECPAs), Anastasiia Gryshana and Rebecca Ravenelle, PhD, in a new Neuronline series. Learn more about Gryshyna and Ravenelle, their research, and their advocacy projects and stay tuned for more ECPA highlights throughout the year.
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Neuroscience QuarterlyPerception — also known as awareness through our senses — has historically been viewed as a static process. We see, hear, smell, and otherwise "read" our surroundings, then process them as sensory inputs. Neuroscientists are discovering, however, that it is not that simple.May 3, 2022
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Advocacy Membership Neuroscience QuarterlyIt is more important than ever that scientists around the globe stand together in support of the future of the scientific enterprise.
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SfN News Training Meetings News from SfNSfN now offers regular peer review training sessions via webinar for SfN members on Neuronline.May 31, 2017
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SfN receives nearly 14,000 abstracts each year for presentation at the annual meeting. After abstract submission, the Program Committee reviews each abstract and sessions them into topically-relevant poster sessions and nanosymposia. A select number of abstracts are assigned to dynamic poster sessions.
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In the mammalian visual system, three functionally distinct parallel processing streams extend from the retina to the visual thalamus and then to the visual cortex: magnocellular (M), parvocellular (P), and koniocellular (K). Tree shrews ( Tupaia belangeri ), a preprimate species, provide an advantageous model to study the K pathway in isolation because, while M and P pathways remain mixed in Lamina 1 (L1), L2, L4, and L5 of the lateral geniculate nucleus (LGN), L3 and L6 receive strictly K-input from the contralateral eye. Additionally, K-input laminae selectively receive glutamatergic axons from the superior colliculus. To reveal how cellular and synaptic properties of K geniculate laminae may differ from M/P laminae and how tectal input may shape the K relay to the cortex, we studied the morphology and connectivity of retinal and tectal terminals in pathway-specific laminae. While confirming that K laminae relay cells contain calbindin, we also found its expression in GABAergic cells across all laminae....Jul 1, 2025
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SfN News Meetings Public Outreach News from SfNRegister now to get the best rates and secure housing for Neuroscience 2018Jul 9, 2018
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Adolescent nicotine use is particularly concerning due to increased susceptibility to long-term effects and dependence during this critical developmental period. This study investigates the therapeutic effects of thiamine on nicotine withdrawal-induced anxiety, anhedonia, and depression in rats. Adolescent rats received nicotine (2 mg/kg, s.c.) for 21 days, followed by 21 days of withdrawal. Thiamine (25 mg/kg or 50 mg/kg, i.p.) was administered during exposure and withdrawal. Behavioral assessments were used to evaluate anxiety- and depressive-like symptoms, and biochemical analyses measured oxidative stress markers, serotonin levels, MAO activity, BDNF, and GFAP as indicators of neuroinflammation in the prefrontal cortex (PFC). Nicotine withdrawal significantly elevated anxiety-, depression-, and anhedonia-like behaviors, increased oxidative stress, and upregulated MAO-A activity and GFAP expression, indicating neuroinflammatory effects. Notably, thiamine administration during both nicotine exposure and ...Aug 8, 2025